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The effect of long-term exposure to kappa-opioid agonists on the ventral tegmental area of the midbrain was studied in mice in vivo. The ventral tegmental area was chosen because it is a major site of opioid action in the brain, and is affected in clinical populations of patients with opioid abuse. Animals were exposed to morphine or diacetylmorphine for seven days (20 mg/kg, s.c., once daily), and microiontophoretic application of glutamate in vitro was used to examine responses of ventral tegmental area neurons to substance P (SP), the main tachykinin neurotransmitter in the spinal cord, and its two primary receptor subtypes, NK-1 and NK-2. Morphine and diacetylmorphine evoked an increase in the number of ventral tegmental area neurons that showed increased responses to SP (107 out of 141), and not to neurokinin A, relative to vehicle-exposed mice
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