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What I did is as follows. I was browsing for 3-9 that apply because. I was actually going for this:
System.Diagnostics.Process process = System.Diagnostics.Process.Start(“”);

When I actually wrote it, it was not working, but after a couple of hours of experimenting I was able to get it running.
There is a bug in the web browser, you actually have to click on the link. I actually right clicked on the link and clicked on “Open in explorer”.
Here is what I came up with:
System.Diagnostics.Process process = System.Diagnostics.Process.Start(@””);

…and it works!
Essentially, you will need to use the process object to access the web browser and then access the link through the Webbrowser…
This is one of those “If you see this, then you are not stupid!” kind of things. 🙂

Interaction between competition and time allocation of work on a smallholder farm in Zimbabwe.
In a farm structure, the role of a farm manager and his position in the farm hierarchy can be based on either personal influence (individual power), or collective power through social authority. The goals of the farm manager are to maximize net income and ensure that work is distributed equitably among those in the farm hierarchy. One way of achieving these goals would be by increasing farm production, consequently, the manager may strive to increase his individual income. The only way of ensuring that work is distributed equitably is by changing time allocation patterns by the farm hierarchy. In this paper we explore the relationship between competing for influence and time allocation patterns. Using the example of a farm that is located in a rural area in Zimbabwe, we analyze a farm’s gross margin (the difference between the expenses and the revenues) and how it is affected by agricultural policy for the period 1994-2004. Using regression analysis, we find that the time allocated to certain production processes in the farm hierarchy is affected by the same group of factors that affect farm management. We find that
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Neuronal ceroid-lipofuscinoses (NCL) are a group of inherited lysosomal storage disorders, characterized by accumulation of autofluorescent storage material. In NCLs, the hexosaminidase A enzyme activity is decreased in childhood, with subsequently severe neurological clinical manifestations, including psychomotor retardation and epilepsy. The aim of this study was to investigate whether the impairment of the guanidinoacetate methyltransferase (GAMT) activity, which is increased in NCLs, is associated with a higher degree of neurodegeneration and the appearance of epilepsy in NCL mice. GAMT enzymatic activity was decreased by 76.5 +/- 7.6% in a mouse model of juvenile neuronal ceroid-lipofuscinoses and by 64.0 +/- 8.4% in a mouse model of late-infantile NCL compared with controls. GAMT expression is increased by approximately 25-fold in the neural tissue of GAMT knockout (KO) mice; however, no significant changes in storage material accumulation and lysosomal membrane enzyme activities were observed in the GAMT KO mice. The results suggest that the inhibition of GAMT may contribute to the clinical manifestation of epilepsy and neuronal degeneration in NCLs..

**Authorship Contributions**

Concept: S.

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